Medical Research Council Clinical Sciences Centre, Hammersmith Hospital, London, and University Department of Psychiatry, Warneford Hospital, Oxford, UK
Clinical Psychopharmacology Unit, University College London, UK
Department of Psychiatry, Yale University, New Haven, Connecticut, USA
Medical Research Council Clinical Sciences Centre and GSK Clinical Imaging Centre, Imperial College London, Hammersmith Hospital, London, UK
Wolfson Molecular Imaging Centre, University of Manchester, UK
University Department of Psychiatry, Warneford Hospital, Oxford, UK
Clinical Psychopharmacology Unit, University College London, UK
Medical Research Council Clinical Sciences Centre, Hammersmith Hospital, London, UK.
Correspondence: Paul Grasby, PET Psychiatry, Cyclotron Building, MRC Clinical Sciences Centre, Imperial College, Hammersmith Hospital, London W12 0NN, UK. Email: paul.grasby{at}csc.mrc.ac.uk
P.G. has received occasional consultancy for GlaxoSmithKline (GSK), MERCK, Pfizer and GE Healthcare. P.C. is an MRC Clinical Scientist. N.V.M. is a GSK employee and holds GSK shares. Z.B. has served on the speakers panel of BMS, AstraZenaca and Janssen.
This study was supported by the Medical Research Council (MRC). R.H. was supported by an MRC studentship.
Background
Animal experimental studies have prompted concerns that widespread use of 3,4-methylenedioxymethamphetamine (MDMA; ecstasy) by young people may pose a major public health problem in terms of persistent serotonin neurotoxicity.
Aims
To determine the status of brain serotonin neurons in a group of abstinent MDMA users.
Method
We assessed the integrity of brain serotonin neurons by measuring serotonin transporter (SERT) binding using positron emission tomography (PET) and [11C]DASB in 12 former MDMA users, 9 polydrug users who had never taken MDMA and 19 controls who reported no history of illicit drug use.
Results
There was no significant difference in the binding potential of [11C]DASB between the groups in any of the brain regions examined.
Conclusions
To the extent that [11C]DASB binding provides an index of the integrity of serotonin neurons, our findings suggest that MDMA use may not result in long-term damage to serotonin neurons when used recreationally in humans.
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