Melbourne Neuropsychiatry Centre, Department of Psychiatry, and Department of Psychology, School of Behavioural Science, Faculty of Medicine, Dentistry and Health Sciences, University of Melbourne, Australia
Melbourne Neuropsychiatry Centre, Department of Psychiatry, and ORYGEN Research Centre, University of Melbourne, Australia
Melbourne Neuropsychiatry Centre, Department of Psychiatry, University of Melbourne, Australia
ORYGEN Research Centre, Department of Psychiatry, University of Melbourne, Australia, and Department of Psychiatry and Psychotherapy, University of Cologne, Germany
ORYGEN Research Centre, Department of Psychiatry, University of Melbourne, Australia
Department of Electrical & Electronic Engineering and Howard Florey Institute, University of Melbourne, Australia
Melbourne Neuropsychiatry Centre, Department of Psychiatry, University of Melbourne, Australia
Department of Psychology, School of Behavioural Science, Faculty of Medicine, Dentistry and Health Sciences, University of Melbourne, Australia
ORYGEN Research Centre, Department of Psychiatry, University of Melbourne, Australia
Melbourne Neuropsychiatry Centre, Department of Psychiatry, and Howard Florey Institute University of Melbourne, Australia
Correspondence: Dr Alex Fornito, Melbourne Neuropsychiatry Centre, Levels 2 and 3, National Neuroscience Facility, 161 Barry Street, Carlton South, VIC 3053, Australia. Email: fornitoa{at}unimelb.edu.au
This research was supported by the Melbourne Neuropsychiatry Centre (Sunshine Hospital), Department of Psychiatry, The University of Melbourne; the National Health and Medical Research Council (NHMRC) (ID 236175; 350241); and the Ian Potter Foundation. Neuroimaging analysis was facilitated by the Neuropsychiatry Imaging Laboratory managed by Ms Bridget Soulsby at the Melbourne Neuropsychiatry Centre and supported by Neurosciences Victoria. S.J.W. was supported by a NHMRC Clinical Career Development Award and a NARSAD Young Investigator Award. A.F. was supported by a JN Peters Fellowship and a NHMRC CJ Martin Fellowship (ID 454797).
Background
The anterior cingulate cortex is frequently implicated in the pathophysiology of bipolar disorder, but magnetic resonance imaging (MRI) studies have reported variable findings owing to a reliance on patient samples with chronic illness and to limited appreciation of the regions heterogeneity.
Aims
To characterise anterior cingulate cortex abnormalities in patients with bipolar disorder experiencing their first episode of psychosis while accounting for regional anatomical variability.
Method
Grey matter volume, surface area and cortical thickness were measured in six anterior cingulate cortex subregions per hemisphere using MRI scans acquired from 26 patients with bipolar I disorder experiencing first-episode psychosis and 26 healthy controls matched for age, gender and regional morphological variability.
Results
Relative to controls, male patients displayed increased thickness in the right subcallosal limbic anterior cingulate cortex. No significant differences were identified in females for grey matter volume or surface area measures. The findings were not attributable to medication effects.
Conclusions
These data suggest that first-episode psychosis in bipolar disorder is associated with a gender-specific, right-lateralised thickness increase in anterior cingulate cortex subregions known to play a role in regulating physiological stress responses.
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