The British Journal of Psychiatry (2002) 180: 300-306
© 2002 The Royal College of Psychiatrists
Protean nature of mass sociogenic illness
From possessed nuns to chemical and biological terrorism fears
ROBERT E. BARTHOLOMEW, PhD
Formerly with the Department of Sociology, James Cook University,
Queensland, Australia
SIMON WESSELY, PhD
Academic Department of Psychological Medicine, King's College School of
Medicine & Institute of Psychiatry, London, UK
Correspondence: S. Wessely, Academic Department of Psychological Medicine, King's College
School of Medicine and Institute of Psychiatry, 103 Denmark Hill, London SE5
8AF, UK
Declaration of interest None.

ABSTRACT
Background Episodes of mass sociogenic illness are becoming
increasingly recognised as a significant health and social problem
that is
more common than is presently reported.
Aims To provide historical continuity with contemporary episodes of
mass sociogenic illness in order to gain a broader transcultural and
transhistorical understanding of this complex, protean phenomenon.
Method Literature survey to identify historical trends.
Results Mass sociogenic illness mirrors prominent social concerns,
changing in relation to context and circumstance. Prior to 1900, reports are
dominated by episodes of motor symptoms typified by dissociation, histrionics
and psychomotor agitation incubated in an environment of preexisting tension.
Twentieth-century reports feature anxiety symptoms that are triggered by
sudden exposure to an anxiety-generating agent, most commonly an innocuous
odour or food poisoning rumours. From the early 1980s to the present there has
been an increasing presence of chemical and biological terrorism themes,
climaxing in a sudden shift since the 11 September 2001 terrorist attacks in
the USA.
Conclusions A broad understanding of the history of mass sociogenic
illness and a knowledge of episode characteristics are useful in the more
rapid recognition and treatment of outbreaks.

INTRODUCTION
Mass sociogenic illness refers to the rapid spread of illness
signs and
symptoms affecting members of a cohesive group, originating
from a nervous
system disturbance involving excitation, loss
or alteration of function,
whereby physical complaints that
are exhibited unconsciously have no
corresponding organic aetiology.
In the standard psychiatric nomenclature,
mass sociogenic illness
is subsumed under the general heading of
somatoform
disorder, subcategorised as conversion
disorder
or hysterical neurosis, conversion type
(
American Psychiatric Association,
1994).

BACKGROUND
Mass sociogenic illness is an under-appreciated social problem
that is both
underreported and often a significant financial
burden to responding emergency
services, public health and
environmental agencies and the affected school or
occupation
site, which is often closed for days or weeks
(
Jones et al, 2000).
The typical study of mass sociogenic illness is written
by health care
professionals who briefly review the contemporary
literature and add a
singular episode in which they were inadvertently
involved. Although hundreds
of books and articles have appeared
on the historical aspects of individual
hysteria (see
Micale, 1995),
excluding the voluminous literature on medieval dance
manias and tarantism,
there is a paucity of books and articles
assessable in English on detailed
historical aspects of mass
sociogenic illness
(
Madden, 1857;
Hirsch, 1883;
Small, 1896;
Burnham, 1924;
Rosen, 1968;
Markush, 1973;
Sirois, 1974;
Bartholomew
& Sirois,
1996,
2000;
Boss, 1997). Given
this
situation, it is easy to lose sight of the dynamic, protean
nature of mass
sociogenic illness and its historical and transcultural
manifestations, which
mirror popular social and cultural preoccupations
that define each era and
reflect unique social beliefs about
the nature of the world.
Wessely (1987) identifies
two types of mass sociogenic illness mass anxiety
hysteria and mass motor hysteria. The former is of
shorter duration, typically one day, and involves sudden, extreme anxiety
following the perception of a false threat. The second category is typified by
the slow accumulation of pent-up stress, is confined to an intolerable social
setting and is characterised by dissociation, histrionics and alterations in
psychomotor activity (e.g. shaking, twitching, contractures), usually
persisting for weeks or months.

THE MIDDLE AGES
Prior to the 20th century, most reports of mass sociogenic illness
involved
motor hysteria incubated by exposure to longstanding
religious, academic or
capitalist discipline. Between the 15th
and 19th centuries, exceedingly strict
Christian religious
orders appeared in some European convents. Coupled with a
popular
belief in witches and demons, this situation triggered dozens
of
epidemic motor hysteria outbreaks among nuns, who were widely
believed to have
been demonically possessed. Episodes typically
lasted months and in several
instances were endured in a waxing
and waning fashion for years. Histrionics
and role-playing
were a significant part of the syndrome. Young girls
typically
were coerced by elders into joining these socially isolating
religious orders, practising rigid discipline in confined, all-female
living
quarters. Their plight included forced vows of chastity
and poverty. Many
endured bland near-starvation diets, repetitious
prayer rituals and lengthy
fasting intervals. Punishment for
even minor transgression included flogging
and incarceration.
The hysterical fits appeared under the strictest
administrators.
Priests were summoned to exorcise the demons, and disliked
individuals often were accused of casting spells and were banished,
imprisoned
or burned at the stake. Witchcraft accusations also
were a way to settle
social and political scores under the
guise of religion and justice. These
rebellious nuns used foul
and blasphemous language and engaged in lewd
behaviour: exposing
genitalia, rubbing private parts or thrusting hips to
denote
mock intercourse (
Calmeil,
1845;
Garnier,
1895;
Loredan,
1912).
Community members often attended the spectacles in a
daily
theatre-like atmosphere while priests would try to exorcise
the demons. An
out-break was recorded in the USA at an Ohio
convent as recently as 1880
(
Davy, 1880).
The number and descriptions of these complex episodes of demon possession
in nunneries are remarkable. There are more than 100 books alone on the
outbreaks at Loudun, France, between 1632 and 1634, where Father Urbain
Grandier purportedly bewitched a convent into hysterical fits and was burned
alive (Huxley, 1952; de Certeau, 1970). On rare
occasions, nuns were executed for bewitching other members of their religious
orders. In 1749, in one of the last recorded cases of its kind, abnormal
movements and trance states affected the Unterzell convent near Würzburg,
Germany. Suspicion of witchcraft fell on a Sister Maria von Mossau who was
beheaded (Robbins, 1966).
Major convent outbreaks were recorded in Lyons in 1526, Wertet in 1550,
Kintorp in 1552, Cologne and Flanders in 1560, Oderheim in 1577, Mons in 1585,
Milan in 1590, Aix in 1609, Lille in 1613, Madrid in 1628, Chinon in 1640,
Louviers in 1642, Auxonne in 1662 and Toulouse in 1681
(Calmeil, 1845;
Madden 1857;
Robbins, 1966). At Cambrai,
France, in 1491 a group of nuns exhibited fits, yelped like dogs and foretold
the future, and in Xante, Spain, in 1560 nuns bleated like sheep, tore
off their veils [and] had convulsions in church
(Robbins, 1966: p. 393). At
one French convent, the nuns meowed together every day at a certain
time for several hours together
(Hecker, 1844: p. 127). During
this period it was widely believed that humans could be possessed by certain
animals considered to be potential demonic familiars, and in France cats were
despised for this reason (Darnton,
1984), possibly explaining the meowing nuns. The
recipe for these outbreaks seems to have been long-standing anxiety, which
engendered dissociation and hyper-suggestibility with the content of
their delusions reflecting the Zeitgeist.
In modern-day Malaysia, under similar conditions, outbreaks of motor
hysteria affect adolescent Muslims sent by their parents or guardians to
socially isolated all-female religious boarding schools. One episode in the
remote state of Kedah affected 36 girls over a period of 5 years. Native
healers (bomohs) were summoned intermittently to exorcise demons.
Symptoms included crying fits, screaming, abnormal movements, possession
states and histrionics. The battle with autocratic administrators climaxed in
1987 when, during an outbreak, the desperate girls took hostages at
knife-point and demanded changes. No one was hurt and the girls, claiming
impunity through possession, were not held legally accountable. The episode
ended after an ex-Prime Minister met with the girls and oversaw their transfer
to a liberal school (Bartholomew,
2000: pp. 192-193).

THE 18th TO THE EARLY 20th CENTURY
During the 18th, 19th and early 20th centuries and the realisation
of the
industrial revolution, harsh working conditions and
weak or non-existent
labour unions led to mass motor hysteria
outbreaks in oppressive Western job
settings, typically factories.
Episodes were recorded in England, France,
Germany, Italy and
Russia and included convulsions
(
Franchini, 1947), abnormal
movements (
Bouzol, 1884) and
neurological complaints (
Schatalow,
1891;
Bekhtereff,
1914). The industrial revolution was notorious
for child labour,
low wages and appalling conditions. The first
recorded outbreak in a job
setting occurred in England at a
Lancashire cotton mill in February 1787,
involving violent
convulsions and sensations of suffocation among one male and
23 female workers (
St Clare,
1787). The episode occurred 2
years after Edmund Cartwright
invented the power-loom, revolutionising
the textile industry
(
Sirois, 1982). The absence of
similar
motor hysteria reports in Western countries during the second
half of
the 20th century may result from union gains and more
rigorous occupational
health and safety regulations. The disappearance
of reports in the former
Soviet Union may reflect the rise
of anti-capitalist and, more recently,
Western-type political
systems
(
Bartholomew & Sirois,
2000).
During this same period strict academic discipline in many European
schools, especially Germany, Switzerland and France, triggered outbreaks of
motor hysteria involving convulsions
(Armainguad, 1879; Hagenbach, 1893), contractures
(Regnard & Simon, 1887), trembling (Laquer, 1888;
Wichmann, 1890) and laughing
(Rembold, 1893). In 1893, a
girls' school in Basel, Switzerland, was affected by contagious shaking and
convulsions involving female students who were unable to complete in-school
written assignments. Symptoms subsided after school hours, relapsing only upon
re-entering school grounds (Aemmer,
1893). In 1904, the same school reported a similar outbreak
(Zollinger, 1906). At
Gross-tinz, Germany, between 28 June and mid-October 1892, hand tremors
affected the entire body and 8/20 victims exhibited altered consciousness and
amnesia (Hirt, 1893). At a
school in Chemnitz, Germany, in February 1906, arm and hand tremors in female
elementary students appeared during their writing exercise hour. The symptoms
began in two pupils but gradually spread to 21 females over 4 weeks
(Schoedel, 1906). The pupils
performed all other manual tasks normally, including gymnastics class.
Electric shocks were administered to those affected, and during their writing
period demanding drills in mental arithmetic were given; the symptoms ceased
soon after.
Some school episodes during this period appear to have been relatively
minor, short-lived and unrelated to academic discipline
(Small, 1896), such as left
arm paralysis in four girls at a London school in February 1907. A girl with
infantile palsy of the left arm fractured her right arm. She returned to class
several weeks later and within a few days three children had lost the
use of their left arms, and a fourth... had such severe pains in her left arm
that she held it to the side and could not be persuaded to use it
(Kerr, 1907: p. 32).
During the 20th century, epidemic hysteria episodes were dominated by
environmental concerns over food, air and water quality, especially
exaggerated or imaginary fears involving mysterious odours. Outbreaks had a
rapid onset and recovery and involved anxiety hysteria. Unsubstantiated claims
of strange odours and gassings were a common contemporary trigger of MSI
outbreaks in schools (Philen et
al, 1989; Selden,
1989; Cole, 1990;
Krug, 1992;
Taylor & Werbicki, 1993;
Small et al, 1994). A
typical incident occurred in August 1985, when 65 students and a teacher at a
Singaporean secondary school were suddenly stricken with chills, headaches,
nausea and breathlessness. A battery of environmental and medical tests were
negative. The episode began when several pupils detected an unusual smell, and
occurred amid a preexisting rumour that a gas had infiltrated the school from
a nearby construction site. Investigators found that those who accepted
the idea succumbed, and those who were indifferent to it were immune
(Goh, 1987: p. 269). This
report is similar to a mystery gas at a Hong Kong school a few years earlier,
affecting over 355 students aged 6-14 years. Before the outbreak there were
rumours of a recent toxic gas scare at a nearby school. Several teachers had
even discussed the incident with their pupils some to the point of
advising them on what action to take if it should hit their school
(Tam et al,
1982).
On 8 July 1972 in Hazelrigg, England, stench from a pigsty may have
triggered an outbreak of stomach pain, nausea, faintness and headache at a
schoolchildren's gala (Smith &
Eastham, 1973). That same year, headache and overbreathing
affecting 16 pupils at a school in Tokyo, Japan, was traced to a pungent smog
(Araki & Honma, 1986). A
1994 episode of breathing problems among 23 students in a female dormitory at
an Arab school in the United Arab Emirates was triggered by a toxic
fire that turned out to be the harmless smell of incense
(Amin et al, 1997).
The perceived threatening agent must be seen as credible to the affected
group. On any given school day, a fainting student would not be expected to
trigger mass sociogenic illness. Yet, if this occurred during the 1991 Persian
Gulf war, and it coincided with the detection of a strange odour in the
building, many of the native school-children might exhibit sudden, extreme
anxiety after assuming that it was an Iraqi poison gas attack. A similar
episode was reported at a Rhode Island elementary school during the Gulf War,
coinciding with intense publicity about chemical weapons attacks on Israel and
the possibility of terrorist attacks on the USA
(Rockney & Lemke,
1992).
Strange odours also were a common 20th century trigger of epidemic anxiety
hysteria in job settings (Colligan &
Murphy, 1979; Boxer et
al, 1984; Boxer,
1985), with environmental pollutant fears leading to lost
productivity time from data processing centres
(Stahl & Lebedun, 1974;
Stahl, 1982) to telephone
offices (Alexander & Fedoruk,
1986), electronic assembly plants
(Colligan et al,
1979) and a compressor factory
(Sinks et al, 1989). An outbreak of breathing problems in male military recruits at their
California army barracks in 1988 happened when the air was laden with a heavy
odour from brush fires and mistaken for toxic fumes. A chance event combined
to worsen the situation. Some recruits were resuscitated in the
early confusion because medics had wrongly assessed their conditions to have
been more serious. These factors created more anxiety and further breathing
problems. A study of the incident showed that those seeing the
resuscitations or witnessing others exhibit symptoms were three
times more likely to report symptoms
(Struewing & Gray,
1990).

CHEMICAL AND BIOLOGICAL WARFARE
During the 20th century, strange odours and the presumed presence
of toxic
gases also were commonly blamed in episodes of mass
hysteria that spread to
communities (
Johnson, 1945;
McLeod, 1975;
Christophers, 1982;
Gamino et al, 1989;
David & Wessely, 1995;
Radovanovic, 1995),
occasionally involving
the fear of chemical and biological weapons. On 22
April 1915,
German soldiers released chlorine gas near Ypres, Belgium, killing
5000 allied troops and injuring 10 000. Before the First World
War ended 90
000 people on both sides were killed by poison
gases and over one million were
injured (
Harris & Paxman,
1991).
The psychological effects of what historian Elvira Fradkin
(
1934) termed the
poison gas scare would haunt
the American psyche for the next three
decades and trigger
several prominent episodes of mass sociogenic illness and
related
social delusions. In rural Virginia between 1933 and 1934 there
were
dozens of reported attacks involving someone spraying
a noxious gas inside
homes at night. After committing significant
time and resources, authorities
concluded that all cases had
mundane origins from backed up chimney
flues to passing
flatulence (
Bartholomew
& Wessely, 1999). Another mad
gasser scare
occurred in Mattoon, Illinois, in 1944
and this also was attributed to anxiety
and imagination (
Johnson,
1945).
Typical symptoms in both episodes included breathlessness,
nausea, headache, dizziness and weakness. Even the famous Martian
invasion
scare on Halloween eve 1938 reflected the preoccupation
with chemical and
biological weapons. Of a survey of listeners
who were frightened or panicked,
20% assumed that the Martian
gas raids were in fact a German gas
attack on
the USA. One typical respondent stated: The announcer
said a
meteor had fallen from Mars and I was sure that he thought
that, but in the
back of my head I had the idea that the meteor
was just a camouflage... and
the Germans were attacking us
with gas bombs
(
Cantril, 1947: p. 160).
There has been a recurrence of this trend since the early 1980s. In March
and April 1983, 947 residents of the Jordan West Bank reported various
psychogenic complaints: fainting, headache, abdominal pain, dizziness
(Modan et al, 1983).
The episode happened amid poison gas rumours and a long-standing Palestinian
mistrust of Jews. Symptoms appeared over 15 days amid rumours and publicity
that poison gas was being sporadically targeted at Palestinians. The outbreak
began in, and was mainly confined to, schools in several adjacent villages. In
one incident, 64 residents in Jenin were rushed to doctors after erroneously
believing that they had been poisoned when thick smoke belched from an
apparently faulty exhaust system on a passing car. Following negative medical
tests, it was evident that no gassings had occurred, the hypothesis was
discredited and the transient symptoms rapidly ceased. A similar episode
occurred in Soviet Georgia during political unrest in 1989. Symptoms spread
among 400 adolescent females at several nearby schools. The incident
transpired after rumours that students were exposed to poison gas by Russian
authorities who had recently used the chemical agent chloropicrin to disperse
an opposition rally (Goldsmith,
1989). Intense media publicity surrounding the confirmed use of
poison gases, and rumours that the students had been gassed, triggered the
rapid spread of anxiety reactions. The transient complaints mimicked the
poison gas symptoms: stomach ache, burning eyes, skin irritation and dry
throat. Media coverage of this and the previous case were instrumental in
spreading both episodes to the wider community. Mass sociogenic illness
flourishes where the threat has a basis in reality. The 1995 terrorist attacks
using sarin nerve gas on the Tokyo subway system by the Aum Shinrikyo sect
triggered a series of MSI episodes involving benign odours
(Wessely, 1995).
Although neither the Serbs nor the Israelis have used chemical and
biological weapons, the bitter and radical nature of the conflicts means that
the belief was congruent with the reality of the threat. Now that the American
people have vivid proof that attacks with chemical and biological weapons are
not science fiction, we are recreating the exact situations that existed in
Kosovo or on the West Bank.

THE 21st CENTURY
The psychological impact of terrorism involves the overexaggerated
response
to a real or perceived terrorist threat. The 11 September
attacks on the USA
and the subsequent use of anthrax as a weapon
have created a heightened state
of anxiety and alertness. At
a time when we are understandably preoccupied
with the threat
from biological and chemical terrorism, an awareness of the
acute physiological disturbances that are associated with, and
sometimes hard
to distinguish from, that threat is more needed
than ever. For instance,
during the Persian Gulf war the first
missile attack on Israel by Iraq was
widely feared to contain
chemical weapons. Although such fears were unfounded,
about
40% of civilians in the immediate vicinity of the attack reported
breathing problems (
Carmeli et
al, 1991).
The social, psychological and economic impact of mass sociogenic illness
and associated anxiety may be as severe as that from confirmed attacks
(Hyams et al, 2002).
For instance, anthrax is not a very effective method for causing mass physical
casualties, yet its mere presence can terrorise a nation and expend a high
toll in human and financial resources. There have been reports of mass
sociogenic illness related to such fears
(Durbin & Vogt, 2001; Villanueva et al,
2001). In one incident a man sprayed a mysterious substance into a
Maryland subway station, resulting in 35 persons being treated for nausea,
headache and sore throats. The fluid later was identified as a relatively
harmless window cleaner (Lellman,
2001). In the Los Angeles subway, a strange odour forced its
temporary closure after many commuters reported feeling ill
(Becerra & Malnic,
2001).
Over 2300 anthrax false alarms occurred during the first 2 weeks of October
2001 (Cable News Network special report, A. Brown, 16 October 2001), many
involving sociogenic symptoms. In one case, a teacher and student reported
minor forearm chemical burns after opening a letter and
discerning a powder in the air. Subsequent analysis revealed no foreign
substance in the envelope (Lehman,
2001). There is a danger of responding to every incident in space
suits and inadvertently amplifying psychological responses. Indeed, the US
government may line the Washington, DC subway system with chemical warfare
agent detectors, yet such devices tend to indicate false alarms. There were
4500 false positives in the Persian Gulf war without a single
confirmed attack. Installation of such alarms may cause disruptions to
transport systems, creating more of an impact than an actual event
(Wessely et al,
2001).
There is concern that after a chemical, biological or nuclear attack,
public health facilities may be rapidly overwhelmed by the anxious and not
just the medical and psychological casualties. Following the Brazilian
Goiania incident, where inadvertent exposure of radiation caused
four deaths and several hundred casualties, about 10 000 people or 10% of the
local population sought medical examinations
(Petterson, 1998). Somatic
symptoms are common in all populations and are more frequent under stressful
conditions (Barsky & Borus,
1999). Although 39% of those exposed during the 1996 Sea
Empress oil spill off Wales reported one or more symptoms, so also did
20% of the unexposed controls (Lyons
et al, 1999). Uncertainty and fear after disasters
commonly generate psychogenic symptoms such as hyperventilation, headache and
nausea, which may be difficult to distinguish from the early stages of a
chemical, biological or nuclear attack. About 4000 of a total 10 000 New York
firefighters who have visited the site of the World Trade Center attacks have
reported respiratory difficulties, dubbed World Trade Center
syndrome. Many others who live and work near ground zero in lower
Manhattan are reporting similar symptoms (shortness of breath, chest pressure
and pain, coughing and general anxiety), despite the New York Health
Department's continuous monitoring of airborne contaminants by city, state and
federal agencies, which continue to indicate contaminant levels below that
which poses a public health threat (Price,
2001).

IS THERE A PREDISPOSITION TO MASS SOCIOGENIC ILLNESS
Scientists typically search for the causes of mass sociogenic
illness by
seeking abnormalities in those affected. Their conflicting
and inconclusive
findings are not surprising because episodes
involve social realities and the
consequences of beliefs. Investigators
of modern-day outbreaks of mass
sociogenic illness in school
and job settings have used standardised
personality tests to
identify social, psychological and even physical
characteristics,
such as gender, in trying to tell why some members of the
same
group are affected whereas others are not. There is no consistent
pattern. Thirty-five affected workers at a fish packaging plant
scored higher
than controls on the Eysenck Personality Inventory
scale for extroversion
(
Smith et al, 1978),
whereas 90 affected
electronics assembly workers scored lower than those who
were
unaffected. Goldberg associated absenteeism and mass sociogenic
illness
(
Goldberg, 1973), but Cole
(
1990) did not. Some results
suggest that those affected score higher on scales for paranoia
(
Goldberg, 1973), neuroticism
(
McEvedy et al, 1966;
Moss & McEvedy, 1966)
and
hysterical traits (
Knight et al,
1965),
whereas others found no correlations
(
Olson, 1928;
Olczak et al, 1971;
Teoh et al, 1975;
Tam et al, 1982).
Gary Small
and his colleagues link academic performance and becoming ill
(
Small et al, 1991),
whereas Goh (
1987) found no
association.
Small also correlated the death of a significant other during
early childhood and being stricken with epidemic hysteria
(
Small & Nicholi, 1982),
and yet this observation was not confirmed
in another study by the same
researcher (
Small & Borus,
1983).
Some investigators report that those affected have
below-average
IQs (
Knight et al,
1965), whereas opposite impressions were
given by others
(
Olson, 1928;
Schuler & Parenton, 1943).
It seems clear that there is no particular predisposition to
mass sociogenic
illness and it is a behavioural reaction that
anyone can show in the right
circumstances.

CONCLUSIONS
A prompt diagnosis of mass sociogenic illness is problematic
because
controversy often surrounds outbreaks and time is needed
to analyse
environmental and medical test results. It has been
argued that rapidly
dissipating, volatile airborne organic
compounds
(
Black & Murray, 2000;
Goode, 2000;
Miller & Ashford, 2000),
or a mixture of low levels of industrial
air pollutants, coupled with
incomplete environmental investigations
(
Faust & Brilliant, 1981),
could have triggered short-lived
symptoms erroneously attributed to mass
sociogenic illness.
Some researchers conclude that sick building syndrome is
attributable,
in whole or part, to polluted air
(
Bauer et al, 1992;
Ryan & Morrow, 1992).
Indeed, a cursory environmental probe
leading to the diagnosis of mass
sociogenic illness among a
group of mostly female garment-makers in Puerto
Rico was later
traced to toxic fumes that had caused respiratory and
degenerative
diseases, and some deaths
(
Cruz, 1990). Hamilton
concluded
that epidemic hysteria at a rayon plant in the
1930s
was actually caused by carbon disulphide exposure
(
Hamilton, 1943).
An outbreak
of abdominal pain, nausea and vomiting at
a British school in 1990 included
classic features of mass
sociogenic illness: a high female attack rate, rapid
onset
and recovery, hyperventilation and line of sight transmission.
Tests
later revealed cucumber pesticide contamination
(
Aldous et al,
1994).
It may be advisable to close the school or job site until negative results
are returned. Closure also should assist in reducing anxiety levels,
temporarily dispersing the group and limiting the potential spread of
symptoms. This will allow time for investigators to determine, in depth,
whether most or all of the eight characteristic features (a combination of
symptoms and conditions) of mass sociogenic illness are present. These are:
symptoms with no plausible organic basis; symptoms that are transient and
benign; symptoms with rapid onset and recovery; occurrence in a segregated
group; the presence of extraordinary anxiety; symptoms are spread via sight,
sound or oral communication; this spread occurs down the age-scale, beginning
in older or higher-status students; and there is a preponderance of female
participants. The issue of diagnosis of mass sociogenic illness is contentious
because it is often viewed as a diagnosis of exclusion. Yet mass sociogenic
illness has distinct features, the confluence of which typically indicates the
presence of psychogenic symptoms. Before air, food and water tests are
returned, it is possible to make a preliminary diagnosis based on these eight
criteria. Knowledge of the characteristic features of mass sociogenic illness,
involving either motor or anxiety symptoms, appears useful in the rapid
preliminary diagnosis and hence the potential treatment of outbreaks.
Treatment of mass sociogenic illness involves identifying and eliminating or
reducing the stress-related stimulus perceived.
No one is immune from mass sociogenic illness because humans continually
construct reality and the perceived danger needs only to be plausible in order
to gain acceptance within a particular group and generate anxiety. As we enter
the 21st century, epidemic hysteria again will mirror the times, likely
thriving on the fear and uncertainty from terrorist threats and environmental
concerns. What new forms it will take and when these changes will appear are
beyond our capacity to predict.

Clinical Implications and Limitations
CLINICAL IMPLICATIONS
- There has been a significant historical shift in the presentation of mass
sociogenic illness.
- Anxiety-related phenomena are now more common than motor phenomena.
- Recent experiences of chemical and biological terrorism, and our general
environmental fears, suggest that such episodes will remain common.
LIMITATIONS
- Literature review only.
- Secondary analysis of articles from many disciplines and cultures.
- Organic factors may have been overlooked in any single episode.

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Received for publication November 13, 2001.
Accepted for publication December 12, 2001.
Related articles in BJP:
- Highlights of this issue
- ELIZABETH WALSH
BJP 2002 180: 0.
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