Hostname: page-component-7c8c6479df-94d59 Total loading time: 0 Render date: 2024-03-29T01:34:26.683Z Has data issue: false hasContentIssue false

Psychiatric Illness and Pernicious Anaemia: A Clinical Re-Evaluation

Published online by Cambridge University Press:  08 February 2018

M. D. Eilenberg*
Affiliation:
Bethlem Royal and Maudsley Hospitals

Extract

Addison (1855) in his classical description wrote of the patient's mind “as occasionally wandering” during the final stages of pernicious anaemia. The relative frequency with which different psychiatric syndromes present differs from that recorded by previous writers and is probably due to earlier psychiatric consultation in the present group, though mode of referral may also play a part. Bowman (1935) found organic confusion in 48 per cent., a death rate of 35 per cent. and a red blood cell count of below 4 0 million in 65 per cent. of his 23 cases. Herman, Most and Joliffe (1937) found organic confusion in 35 per cent. compared with the present writer's 5 per cent., a death rate of 22 · 5 per cent. compared with 10 per cent. and a red blood cell count of under 3 · 0 million in 75 per cent. of his 40 cases, compared with 25 per cent. of the present group.

Type
Original Articles
Copyright
Copyright © Royal College of Psychiatrists, 1960 

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

References

Addison, T., London Med. Gaz., 1855, 43, 517.Google Scholar
Arnstein, H. R. V., Vitamin Metabolism (Proc. 4th Int. Cong. of Biochemistry; Ed.: Umbrett, W. and Molitor, H.), 1960. London: Pergamon Press.Google Scholar
Bicknell, F., and Prescott, F., The Vitamins in Medicine, 1953. London: W. Heinemann.Google Scholar
Bowman, K. M., Amer. J. Psych., 1935, 92, 371.Google Scholar
Canterow, A., and Schepartz, B., Biochemistry, 1954. London: W. B. Saunders ' Co. Google Scholar
Qutchley, MacDonald, Lancet, 1931, i, 1119.Google Scholar
Davies, D. L., Brit. J. Soc. Med., 1956, 10, 123.Google Scholar
Earl, C. J., Earl, M. F. S., Hawary, E. L., Thompson, R. H. S., and Webster, G. R., Lancet, 1953, i, 115.Google Scholar
Herman, M., Most, F., and Joliffe, N., Arch. Neurol. and Psych., 1937, 38, 348.Google Scholar
Jewsbury, E. C. D., Lancet, 1954, ii, 307.CrossRefGoogle Scholar
Ling, C. T., and Crow, B. F., J. Biol. Chem., 1953, 202, 445.Google Scholar
Romano, J., and Engel, G. L., Arch. Neurol. and Psych., 1944, 51, 356.Google Scholar
Samson, D. C., Swisher, S. N., Christian, R. M., and Engel, G. L., A.M.A. Arch. Int. Med., 1952, 90, 4.Google Scholar
Scheinberg, P., Blood, 1951, 6, 213.Google Scholar
Strickland, K. P., Guy's Hosp. Rep., 1956, 105, 108.Google Scholar
Walton, J. N., Kiloh, L. G., Osselton, J. W., and Farrall, J., EEG and Clin. Neurophysiol., 1954, 6, 45.Google Scholar
Submit a response

eLetters

No eLetters have been published for this article.