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The Relationship Between 17-Hydroxycorticosteroid Excretion and Glucose Utilization in Depressions

Published online by Cambridge University Press:  29 January 2018

I. G. Pryce
I. G. Pryce*
Affiliation:
Whitchurch Hospital, Cardiff
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Extract

Many authors have observed a decrease in glucose-tolerance (G.T.) in melancholia (sec reviews by McFarland and Goldstein, 1939; Altschule, 1953). Using an intravenous test, the present author confirmed that G.T., and also body weight, were significantly lower in a group of depressions than in a matched control group (Pryce, 1958a), but that within the depressions G.T. was not related to body weight. In a subsequent study also (Pryce, 1958b) change in G.T. after treatment was not related to change in body weight nor to three measures of change in emotional state. These studies, therefore, failed to support the views that the decreased G.T. in depressions is related either to nutritional or to emotional factors.

Type
Research Article
Information
The British Journal of Psychiatry , Volume 110 , Issue 464 , January 1964 , pp. 90 - 94
Copyright
Copyright © Royal College of Psychiatrists, 1964 

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References

Altschule, M. D. (1953). Bodily Physiology in Mental and Emotional Disorders. 1st edition. New York: Grime and Stratton.Google Scholar
Appleby, J. L., Gibson, G., Norymberski, J. K., and Stubbs, R. D. (1955). Biochem. J., 60, 453.CrossRefGoogle Scholar
Board, F., Wadeson, R., and Persky, J. (1957). Arch. Neurol. Psychiat., Chicago, 78, 612.CrossRefGoogle Scholar
Callow, R. K. (1950). Hormone Assay. New York: Academic Press Inc. p. 374.Google Scholar
Cleghorn, R. A., and Curtis, G. C. (1959). Canad. Psychiat. Ass. J., 4, Special Supplement, p. 18.Google Scholar
Cope, C. L., and Black, E. G. (1959). Brit. med. J., ii, 1117.Google Scholar
Duncan, L. J. P. (1956). Quart. J. exp. Physiol., 41, 85.Google Scholar
Froesch, E. R., Winegrad, A. I., Renold, A. E., and Thorn, G. W. (1958). J. Clin. Invest., 37, 524.CrossRefGoogle Scholar
Gibbons, J. L., and McHugh, P. (1962). J. Psychiat. Res., 1, 162.CrossRefGoogle Scholar
Ikkos, R., and Lufi, R. (1957). Acta endoc∗∗∗s., Copenhagen, 25, 312.Google Scholar
King, E. J. (1951). Microanalysis in Medical Biochemistry. London: Churchill, p. 130.Google Scholar
Lingjaerde, O. (1956). Acta Psychiat. kbh. Suppl., 106.Google Scholar
Lundbaek, K. (1948). Tale J. Biol. Med., 20, 533.Google Scholar
McFarland, P. A., and Goldstein, H. (1939). Amer. J. Psychiat., 96, 21.Google Scholar
M.R.C. Committee on Clinical Endocrinology (1951). Lancet, ii, 585.Google Scholar
Nabarro, J. D. N. (1960). Brit. med. J., ii, 553.Google Scholar
Nabarro, J. D. N., Moxham, A., and Walker, G. (1957). Ibid., ii, 1018.Google Scholar
Pryce, I. G. (1958a). J. Ment. Sci., 104, 421.Google Scholar
Pryce, I. G. (1958b). Ibid., 104, 1079.Google Scholar
Pryce, I. G. (1960). M.D. Thesis, University of London Library.Google Scholar
Vestergaard, P., and Leverett, R. (1958). J. Lab. clin. Med., 51, 211.Google Scholar
Weil-Malherbe, N., and Bone, A. D. (1955). J. Ment. Sci., 101, 733.Google Scholar
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