Hostname: page-component-7c8c6479df-xxrs7 Total loading time: 0 Render date: 2024-03-28T13:33:55.767Z Has data issue: false hasContentIssue false

Toxic Psychosis Following Mumps

Published online by Cambridge University Press:  29 January 2018

Kenneth M. G. Keddie*
Affiliation:
Barrow and Glenside Hospitals, Bristol

Extract

Mumps, or epidemic parotitis, is caused by a filterable virus and is characterized by temporary enlargement of the parotid and sometimes other salivary glands and by the occurrence of complications, of which orchitis in adults and in post-pubertal boys is the most common. The virus is pantropic causing central and peripheral nervous complications in many cases. In civilized countries the disease is endemic but local outbreaks occur. One such outbreak occurred in Bristol during 1964, in the spring, the season when mumps is most prevalent. The case that is described here formed a part of that particular epidemic. Before proceeding to the case report the literature on the psychiatric complications of mumps will be discussed.

Type
Research Article
Copyright
Copyright © Royal College of Psychiatrists, 1965 

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

References

Allen, I. N. (1964). Personal communication.Google Scholar
Bowers, D., and Weatherhead, D. S. P. (1953). Canad. med. Ass. J., 69, 4955.Google Scholar
Brain, R. (1960). Clinical Neurology. London: Oxford University Press.Google Scholar
Delage, J. (1955). Laval Mid., 20, 175183. (Abstract in English in Excerpta Medica, Section VIII, 1956, 9, No. 2632.) Google Scholar
Donohue, W. L. (1941). J. Pediat., 19, 4252.Google Scholar
Donohue, W. L., Playfair, F. D., and Whitaker, L. (1955). Ibid., 47, 395.Google Scholar
Eberlein, W. R., and Lynxwiler, C. P. (1947). Ibid., 31, 513.Google Scholar
Fallon, R. J. (1957). J. Roy. Nav. med. Serv., 43, 228231.Google Scholar
Finkelstein, H. (1938). J. Amer. med. Ass., 111, 1719.CrossRefGoogle Scholar
Frankland, A. W. (1941). Brit. med. J., ii, 48.Google Scholar
Kilham, L. (1949). Amer. J. Dis. Child., 78, 324.Google Scholar
Klotz, M. (1925). Handbuch der Inneren Medizin, Berlin: Springer, p. 624.Google Scholar
McKaig, C. B., and Woltman, H. W. (1934). Arch. Neurol. Psychiat. (Chic.), 31, 794.Google Scholar
Mayer-Gross, W., Slater, E., and Roth, M. (1960). Clinical Psychiatry. London: Cassell & Co. Ltd.Google Scholar
Miller, H. G., Stanton, J. B., and Gibbons, J. L. (1956). Quart. J. Med., 49, 427505.Google Scholar
Miller, H. G., Stanton, J. B., and Evans, M. J. (1953). Ibid., 22, 347.Google Scholar
Mitchell, W. S. (1911). Lancet, i, 23.Google Scholar
Phillips, S. (1911). Ibid., i, 23.Google Scholar
Roux, G. C. (1914). Gaz. Hôp. Paris, 87, 549.Google Scholar
Russell, R. R., and Donald, J. C. (1958). Brit. med. J., ii, 2730.CrossRefGoogle Scholar
Smith, R. P. (1889). Lancet, ii, 265.Google Scholar
Tabor, S. H., and Newman, B. (1940). Arch. Pediat., 57, 133.Google Scholar
Urechia, C. I. (1938). Encéphale, 33, 144.Google Scholar
Van Bibber, J. (1885). Med. News, 47, 678.Google Scholar
Submit a response

eLetters

No eLetters have been published for this article.