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Studies of Water Metabolism in Essential Epilepsy

Published online by Cambridge University Press:  08 February 2018

Guy D. Greville ,
Tudor S. G. Jones  and
W. F. G. Hughes
Guy D. Greville
Affiliation:
From the Biochemical Department, Runwell Hospital, near Wickford, Essex
Tudor S. G. Jones
Affiliation:
From the Biochemical Department, Runwell Hospital, near Wickford, Essex
W. F. G. Hughes
Affiliation:
From the Biochemical Department, Runwell Hospital, near Wickford, Essex
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Extract

The comparatively old belief, that the epileptic patient shows a retention of water before the onset of a major seizure and an increase in the water excretion afterwards, achieved great importance from two findings of more recent date : on the one hand, the frequency of the seizures may be increased on hydration of the organism by administration of excessive amounts of water in combination with antidiuretics; on the other, the frequency may be decreased on dehydration either by restriction of the water intake or by other means (see for example Fay, 1929, 1930; McQuarrie, 1929; McQuarrie and Peeler, 1931; McQuarrie et al., 1932; Engel et al., 1934; Clegg and Thorpe, 1935; Stubbe Teglbjaerg, 1936; Ziskind et al., 1939; Hagenmeyer and Langelüddeke, 1939; but contrast Cameron, 1931; Fetterman and Kumin, 1933; Wilson and Limberger, 1933; Stone and Chor, 1937; Pette and Janzen, 1938). It is therefore most desirable that the value of the evidence connecting water balance and the occurrence of spontaneous seizures should be assessed.

Type
Part I.—Original Articles
Information
Journal of Mental Science , Volume 86 , Issue 361 , March 1940 , pp. 195 - 229
Copyright
Copyright © Royal College of Psychiatrists, 1940 

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References

Albrecht, H. (1938), Allgemeine Zeitschr. f. Psychiat. u.i. Grenz., 109, 262.Google Scholar
Allers, R. (1910), Journ. Psychol. Neurol. Leipzig, 16, 157.Google Scholar
Benedict, F. G., and Root, H. F. (1926), Arch. intern. Med., 38, 1.CrossRefGoogle Scholar
Cameron, D. E. (1931), Amer. Journ. Psychiat., 11, 123.CrossRefGoogle Scholar
Clegg, J. L., and Thorpe, F. T. (1935), Lancet, 1, 1381.CrossRefGoogle Scholar
Cole, S. W. (1933), Practical Physiological Chemistry, ninth edition. Cambridge: Heffer.Google Scholar
Contini, M. (1936), Rass. Studi Psichiat., 25, 813; (1937), Zbl. ges. Neurol. Psychiat., 86, 275.Google Scholar
Davies, W. L. (1932), Analyst, 57, 79.CrossRefGoogle Scholar
DeCrinis, M. (1920), Die Beteiligung der humoralen Lebensvorgänge des menschlichen Organismus am epileptischen Anfall. Berlin: Springer.CrossRefGoogle Scholar
DuBois, E. F. (1936), Basal Metabolism in Health and Disease. London: Baillière. Tindall & Cox.Google Scholar
Engel, R., McQuarrie, I., and Ziegler, M. (1934), Arch. exp. Path. Pharmak., 174, 555.CrossRefGoogle Scholar
Fay, T. (1929), Amer. Journ. Psychiat., 8, 783.CrossRefGoogle Scholar
Idem (1930), Arch. Neurol. Psychiat., 23, 920.CrossRefGoogle Scholar
Fetterman, J. L., and Kumin, H. J. (1933), Journ. Amer. med. Assoc., 100, 1005.CrossRefGoogle Scholar
Frisch, F. (1937), Die Epilepsie. Wien, Leipzig and Bonn: Weidmann.Google Scholar
Idem and Fried, E. (1927), Z. ges. exp. Med., 56, 766.Google Scholar
Frisch, F., and Walter, K. (1922), Z. ges. Neurol. Psychiat., 79, 366.CrossRefGoogle Scholar
Frisch, F., and Weinberger, W. (1922), ibid., 79, 576.Google Scholar
Gamble, J. L. (1930), Arch. Neurol. Psychiat., 23, 915.CrossRefGoogle Scholar
Idem and Hamilton, R. (1927), Johns Hopk. Hosp. Bull., 41, 389.Google Scholar
Geller, W. (1936), Klin. Wochenschr., 15, 168.CrossRefGoogle Scholar
Hagenmeyer, L., and Langelüddeke, A. (1939), Z. ges. Neurol. Psychiat., 164, 195.CrossRefGoogle Scholar
Hodskins, M. B., Guthrie, R. H., and Naurison, J. Z. (1932), Amer. Journ. Psychiat., 11, 623.CrossRefGoogle Scholar
Isenschmid, , (1918), Med. Klinik, 14, 1128.Google Scholar
Jackson, and Gillis, (1920), Bureau of Standards Scientific Paper No. 375; cited by C. A. Google Scholar
Mitchell, (1930), Recent Advances in Analytical Chemistry, 1. London: Churchill.Google Scholar
Jones, T. S. G. (1936), Journ. Dairy Res., 7, 41.CrossRefGoogle Scholar
Lavietes, P. H. (1935), Journ. clin. Invest., 14, 57.CrossRefGoogle Scholar
Lennox, W. G., and Cobb, S. (1928), Epilepsy. London: Baillière, Tindall & Cox.Google Scholar
McCance, R. A., and Shipp, H. L. (1933), Spec. Rep. Ser. Med. Res. Coun., Lond., No. 187.Google Scholar
McKenzie, B. W., and McChesney, E. W. (1935), Arch. Neurol. Psychiat., 34, 764.CrossRefGoogle Scholar
McQuarrie, I. (1929), Amer. Journ. Dis. Child., 38, 451.CrossRefGoogle Scholar
Idem , Manchester, R. C., and Husted, C. (1932), ibid., 43, 1519.Google Scholar
McQuarrie, I., and Peeler, D. B. (1931), Journ. clin. Invest., 10, 915.CrossRefGoogle Scholar
Meyer, M., and Brühl, F. (1922), Z. ges. Neurol. Psychiat., 75, 158.CrossRefGoogle Scholar
Newburgh, L. H., Johnston, M. W., Lashmet, F. H., and Sheldon, J. M. (1937), Journ. Nutrit., 13, 203.CrossRefGoogle Scholar
Newburgh, L. H., Johnston, M. W., Wiley, F. H., Sheldon, J. M., and Murrill, W. A. (1937), ibid., 13, 193.Google Scholar
Newburgh, L. H., Wiley, F. H., and Lashmet, F. H. (1931), Journ. clin. Invest., 10, 703.CrossRefGoogle Scholar
Nixon, J. A., and Nixon, D. G. C. (1938), Textbook of Nutrition. Oxford: University Press.Google Scholar
Palmer, W. W. (1918), Journ. biol. Chem., 33, 119.CrossRefGoogle Scholar
Peters, J. P., Kydd, D. M., and Lavietes, P. H. (1933), Journ. clin. Invest., 12, 689.CrossRefGoogle Scholar
Pette, H., and Janzen, R. (1937), Deutsche Zeitschr. f. Nervenheilk., 145, 1.CrossRefGoogle Scholar
Plimmer, R. H. A. (1938), Organic and Biochemistry. London: Longmans, Green.Google Scholar
Rohde, E. (1908), Deutsch. Arch. klin. Med., 95, 148.Google Scholar
Stone, T. T., and Chor, H. (1937), Arch. Neurol. Psychiat., 38, 798.CrossRefGoogle Scholar
Stubbe Teglbjaerg, H. P. (1936), Acta Psychiatrica et Neurologica, Suppt. ix.Google Scholar
West, E. S., Scharles, F. H., and Peterson, V. L. (1929), Journ. biol. Chem., 82, 137.CrossRefGoogle Scholar
Wildermuth, H. (1925), Z. ges. Neurol. Psychiat., 95, 120.CrossRefGoogle Scholar
Wiley, F. H., and Newburgh, L. H. (1931), Journ. clin. Invest., 10, 723.CrossRefGoogle Scholar
Wilson, G., and Limberger, W. A. (1933), Journ. Amer. med. Assoc., 101, 110.CrossRefGoogle Scholar
Wuth, O. (1928), in Bumke's, O. Handbuch der Geisteskrankheiten, 3, pt. 3, 157, 195.Berlin: Springer.Google Scholar
Ziskind, E., Somerfeld-Ziskind, E., and Bolton, R. (1939), Journ. Nerv. and Ment. Dis., 89, 52.CrossRefGoogle Scholar
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