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Compensatory Enlargement of the Posterior Communicating Artery Following Arterio-Sclerotic Changes of the Posterior Cerebral Artery

Published online by Cambridge University Press:  08 February 2018

R. Klein
R. Klein*
Affiliation:
From the Department of Mental Disease Research, Birmingham University
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Extract

While a vast literature refers to anomalies in various arteries of the brain, there are only a few reports concerning anatomical changes of vessels following altered circulatory conditions. Critchley mentions in his study of the anterior cerebral artery and its syndromes, an observation in which an arterio-sclerotic anterior cerebral artery of one side was so small as to be incapable of maintaining an adequate circulation, so that by way of an abnormally large anterior communicating artery the opposite anterior cerebral supplied both hemispheres. Critchley adds to that case several similar observations from the literature. The application of arteriography to circulatory disturbances has directed attention to analogous cases. Thus Moniz reported several observations in which a closure of the carotid artery of one side led to a distension of the anterior communicating in order to establish collateral circulation. The same mechanism appeared to be effective in the following two cases which we met in the course of our studies on brain vessels.

Type
Part I.—Original Articles
Information
Journal of Mental Science , Volume 86 , Issue 365 , November 1940 , pp. 1086 - 1091
Copyright
Copyright © Royal College of Psychiatrists, 1940 

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References

Abbie, A. A. (1933), Brain, 56.CrossRefGoogle Scholar
Idem (1933), Journ. Anat., 67.Google Scholar
Ibid. (1934), 68.Google Scholar
Beadles, C. F. (1907) Brain, 30.Google Scholar
Beevor, C. E. (1907), ibid., 30.Google Scholar
Blachford, E. Fawcett (1906), Journ, Anat., 40.Google Scholar
Critchley, M. (1930), Brain, 53.Google Scholar
Idem (1933), Journ. Anal., 67.Google Scholar
Elvidge, A. R. (1938), Ass. f. Res. in Nerv. and Ment. Dis., Baltimore.Google Scholar
Foix, Ch., and Hillemand, (1935), Encéph., 20.Google Scholar
Idem (1925), Rev. Near., 32.Google Scholar
Hiller, F. (1936), Handb. d. Neurol., II. Berlin: I. Springer.Google Scholar
Krieg, W. (1939), Zbl. f. Chirurgie, 12.Google Scholar
Le Gros Clark, W. E. (1938), The Hypothalamus. London.Google Scholar
Moniz, E. (1938), Abl. f. Neurochir., 3.Google Scholar
Riechert, A. (1938), Nervenarzt., II. Google Scholar
Stopford, J. S. B. (1915), Journ. Anat., 50.Google Scholar
Idem (1916), ibid., 51.Google Scholar
Williams, D. J. (1936), ibid., 59.Google Scholar
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