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Parieto-Occipital Syndrome Following Carbon Monoxide Poisoning

Published online by Cambridge University Press:  08 February 2018

G. D. F. Steele
Affiliation:
Bristol Mental Hospitals
A. B. Hegarty
Affiliation:
Bangour Hospital, West Lothian

Extract

Suicidal attempts by coal-gas poisoning are very common. Carbon monoxide is a principal constituent of this gas, and can produce severe and permanent brain damage. It is surprising, therefore, to find so few cases of chronic organic psychosis attributed to this cause in mental hospital practice. Rosseter (1928) found only one example of permanent psychosis in 2,000 cases of carbon monoxide asphyxiation. Shillito, Drinker and Shaughnessy (1936) made a follow-up study of 21,000 cases of acute carbon monoxide poisoning. In only 43 of these were the after-effects sufficiently severe to warrant their admission to a mental hospital. Twenty-three subsequently recovered, 11 died, and 9 suffered permanent nervous and mental sequelae. They found that the ratio of psychosis following carbon monoxide poisoning to other psychoses was 1 in 2,000. Henderson and Gillespie (1944) could find only one such case in 5,000 consecutive admissions.

Type
Part I.—Original Articles
Copyright
Copyright © Royal College of Psychiatrists, 1950 

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References

Adler, A. (1944), Arch. Neurol. and Psychiat., 51, 243–59.Google Scholar
Brain, R. W. (1941), Brain, 64, 244.Google Scholar
Gerstmann, J. (1940), Arch. Neurol. and Psychiat., 44, 398408.CrossRefGoogle Scholar
Gerstmann, J. (1942), Arch. Neurol. and Psychiat., 48, 890.Google Scholar
Grinker, R. R. (1926), J. Nerv. Ment. Dis., 64, 18.CrossRefGoogle Scholar
Hemphill, R. E., and Klein, R. (1948), J. Ment. Sci., 94, 611–22.Google Scholar
Henderson, D. K., and Gillespie, R. D. (1944), A Textbook of Psychiatry. 6th ed. Google Scholar
Holmes, G. (1918), Brit. J. Ophth., 2, 449506.Google Scholar
Holmes, G. (1919), Brit. Med. J., 2, 230.CrossRefGoogle Scholar
Kleist, K. (1922), Kriegsverletzungen des Gehirns, Leipzig.Google Scholar
Mayer-Gross, W. (1935), Proc. Roy. Soc. Med., 28, 1203.CrossRefGoogle Scholar
Mayer-Gross, W. (1936), Proc. Roy. Soc. Med., 29, 66.Google Scholar
Mayer-Gross, W. (1936), J. Ment. Sci., 88, 744.CrossRefGoogle Scholar
Mott, F. W. (1907), Arch. Neurol. from Pathological Laboratory of London County Asylums, 3.Google Scholar
Neilsen, J. M. (1936), Arch. Neur. and Psychiat., 38, 108–35.Google Scholar
Neilsen, J. M. (1938), Arch. Neur. and Psychiat., 39, 536.Google Scholar
Neilsen, J. M. (1938), Bull. Los. Ang. Neur. Soc., 3, 127.Google Scholar
Neilsen, J. M. (1940), Bull. Los. Ang. Neur. Soc., 3, 135.Google Scholar
Raskin, N., and Mullaney, O. C. (1940), J. Nerv. Ment. Dis., 92, 640–59.CrossRefGoogle Scholar
Riddoch, G. (1917), Brain, 40, 15.Google Scholar
Rossiter, F. S. (1928), Carbon Monoxide Poisoning. Pittsburgh Steel Co.Google Scholar
Sayers, R. R., and Davenport, S. J. (1936), Public Health Bulletin, 195.Google Scholar
Schilder, P., and Isakower, O. (1928), Ztschr. f. d. ges. Neurol. Psychiat., 11, 102–42.Google Scholar
Shillito, H., Drinker, C. K., and Shaughnessy, T. K. (1936), J.A.M.A., 106, 669–74.Google Scholar
Stengel, E. (1929), Zeitsch. ge. Neur. Psychiat., 122, 597.Google Scholar
Stengel, E. (1944), J. Ment. Sci., 90, 753.CrossRefGoogle Scholar
Stewart, R. M. (1920), J. Neurol., and Psychopathol., 1, 105.Google Scholar
Wilson, G., and Winkleman, W. (1922), Arch. Neurol. and Psychiat., 13, 192–6.Google Scholar
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